Download Bacterial Adhesion to Host Tissues: Mechanisms and by Michael Wilson PDF

By Michael Wilson

ISBN-10: 0521801079

ISBN-13: 9780521801072

Because of my evolving learn, this ebook is as well timed because it is easily written. whereas the process taken is predominantly a molecular genetics view to microbial adhesion, the bankruptcy authors & editor have awarded the wealth of data in this subject in a good equipped manner.Each bankruptcy is easily equipped and each idea given is then supported with examples of clinical facts. on the finish of every bankruptcy are pages of references that i've got used to additional my learn (as will somebody else drawn to this topic).For the photos, the editor has performed a great task culling out the simplest. From uncomplicated gene/operon diagrams to electron micrographs to paint plates and images, the authors and editor have judicially supplied merely sound transparent graphics.If you will have a e-book that may supply a molecular method of bacterial adhesion, this can be a good start line. whereas a number of chapters are too constrained of their applicablity to the general resolution of this factor, the remainder greater than make up for those minor shortfalls.Keep in brain, this booklet isn't for the beginner from simply any box of microbiology. even so, nor is the topic. while you're interested by this becoming box of analysis, this booklet can be a useful addition in your library.

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Extra info for Bacterial Adhesion to Host Tissues: Mechanisms and Consequences

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Virji 28 Neisseria meningitidis strains are isolated from the nasopharynx of up to 30% of healthy individuals and may be classified as commensals of the human respiratory tract. Individual strains may produce one of several capsular chemotypes (serogroups), designated: A, B, C, 29E, H, I, K, L, W135, X, Y, and Z (Cartwright, 1995, p. 24). Of these A, B, and C are most commonly encountered during disease. Serogroup A predominates in Africa and is responsible for epidemic spread whereas serogroups B and C prevail in the West and are associated with sporadic outbreaks.

1998a). Three new members of the serine-aspartate repeat protein multigene family of Staphylococcus aureus. Microbiology 144, 3387–3395. , Durussel, I. A. (1998b). The binding of calcium to the B-repeat segment of SdrD, a cell surface protein of Staphylococcus aureus. Journal of Biological Chemistry 273, 31145–31152. , Patti, J. J. (2001) Protection against Staphylococcus aureus arthritis by vaccination with clumping factor A, a novel virulence determinant. Journal of Infectious Diseases, in press.

It can be quantitatively released from cells by treatment with LiCl and is thus not covalently anchored. The Map protein comprises six repeats of a 110 amino acid residue motif with a central portion composed of a subdomain with high homology to the peptide-binding groove of mammalian major histocompatibility complex class II (MHCII) molecules. Map is capable of interacting with a variety of proteins and peptides, including many ECM proteins of the host. , 1999). , 1981). Biofilms comprise multiple layers of cells embedded in an amorphous extracellular glycocalyx.

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